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DAF in diabetic patients is subject to glycation/inactivation at its active site residues()
Decay accelerating factor (DAF or CD55) is a cell associated C3 and C5 convertase regulator originally described in terms of protection of self-cells from systemic complement but now known to modulate adaptive T cell responses. It is expressed on all cell types. We investigated whether nonenzymatic...
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| Foilsithe in: | Mol Immunol |
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| Main Authors: | , , , , , |
| Formáid: | Artigo |
| Teanga: | Inglês |
| Foilsithe: |
2017
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| Ábhair: | |
| Rochtain Ar Líne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5884443/ https://ncbi.nlm.nih.gov/pubmed/28886871 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.molimm.2017.06.036 |
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