Minimal SPI1-T3SS effector requirement for Salmonella enterocyte invasion and intracellular proliferation in vivo

Effector molecules translocated by the Salmonella pathogenicity island (SPI)1-encoded type 3 secretion system (T3SS) critically contribute to the pathogenesis of human Salmonella infection. They facilitate internalization by non-phagocytic enterocytes rendering the intestinal epithelium an entry sit...

Täydet tiedot

Tallennettuna:
Bibliografiset tiedot
Julkaisussa:PLoS Pathog
Päätekijät: Zhang, Kaiyi, Riba, Ambre, Nietschke, Monika, Torow, Natalia, Repnik, Urska, Pütz, Andreas, Fulde, Marcus, Dupont, Aline, Hensel, Michael, Hornef, Mathias
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Public Library of Science 2018
Aiheet:
Research Article
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC5862521/
https://ncbi.nlm.nih.gov/pubmed/29522566
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.ppat.1006925
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