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RIPK3-deficient mice were not protected from nephrotoxic nephritis
BACKGROUND: Necrotizing glomerular lesions are a feature of severe glomerulonephritis. Unlike apoptosis, cellular necrosis has the potential to release damage-associated proteins into the microenvironment, thereby potentiating inflammation. Until recently necrosis was thought to be an unregulated ce...
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| Publicat a: | BMC Nephrol |
|---|---|
| Autors principals: | , , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
BioMed Central
2018
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5853157/ https://ncbi.nlm.nih.gov/pubmed/29540162 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s12882-018-0850-4 |
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