SEMA3C drives cancer growth by transactivating multiple receptor tyrosine kinases via Plexin B1

Growth factor receptor tyrosine kinase (RTK) pathway activation is a key mechanism for mediating cancer growth, survival, and treatment resistance. Cognate ligands play crucial roles in autocrine or paracrine stimulation of these RTK pathways. Here, we show SEMA3C drives activation of multiple RTKs...

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Detalhes bibliográficos
Publicado no:EMBO Mol Med
Main Authors: Peacock, James W, Takeuchi, Ario, Hayashi, Norihiro, Liu, Liangliang, Tam, Kevin J, Al Nakouzi, Nader, Khazamipour, Nastaran, Tombe, Tabitha, Dejima, Takashi, Lee, Kevin CK, Shiota, Masaki, Thaper, Daksh, Lee, Wilson CW, Hui, Daniel HF, Kuruma, Hidetoshi, Ivanova, Larissa, Yenki, Parvin, Jiao, Ivy ZF, Khosravi, Shahram, Mui, Alice L‐F, Fazli, Ladan, Zoubeidi, Amina, Daugaard, Mads, Gleave, Martin E, Ong, Christopher J
Formato: Artigo
Idioma:Inglês
Publicado em: John Wiley and Sons Inc. 2018
Assuntos:
Research Articles
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5801490/
https://ncbi.nlm.nih.gov/pubmed/29348142
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.15252/emmm.201707689
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