Frataxin‐deficient neurons and mice models of Friedreich ataxia are improved by TAT‐MTScs‐FXN treatment

Friedreich ataxia (FA) is a rare disease caused by deficiency of frataxin, a mitochondrial protein. As there is no cure available for this disease, many strategies have been developed to reduce the deleterious effects of such deficiency. One of these approaches is based on delivering frataxin to the...

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Bibliografske podrobnosti
izdano v:J Cell Mol Med
Main Authors: Britti, Elena, Delaspre, Fabien, Feldman, Anat, Osborne, Melissa, Greif, Hagar, Tamarit, Jordi, Ros, Joaquim
Format: Artigo
Jezik:Inglês
Izdano: John Wiley and Sons Inc. 2017
Teme:
Original Articles
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC5783845/
https://ncbi.nlm.nih.gov/pubmed/28980774
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1111/jcmm.13365
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