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Sodium Channel Remodeling in Subcellular Microdomains of Murine Failing Cardiomyocytes
BACKGROUND: Cardiac sodium channel (NaV1.5) dysfunction contributes to arrhythmogenesis during pathophysiological conditions. Nav1.5 localizes to distinct subcellular microdomains within the cardiomyocyte, where it associates with region‐specific proteins, yielding complexes whose function is locati...
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| Publicado no: | J Am Heart Assoc |
|---|---|
| Main Authors: | , , , , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
John Wiley and Sons Inc.
2017
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5779058/ https://ncbi.nlm.nih.gov/pubmed/29222390 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/JAHA.117.007622 |
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