Receptor for advanced glycation end products mediates sepsis-triggered amyloid-β accumulation, Tau phosphorylation, and cognitive impairment

Patients recovering from sepsis have higher rates of CNS morbidities associated with long-lasting impairment of cognitive functions, including neurodegenerative diseases. However, the molecular etiology of these sepsis-induced impairments is unclear. Here, we investigated the role of the receptor fo...

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Vydáno v:J Biol Chem
Hlavní autoři: Gasparotto, Juciano, Girardi, Carolina S., Somensi, Nauana, Ribeiro, Camila T., Moreira, José C. F., Michels, Monique, Sonai, Beatriz, Rocha, Mariane, Steckert, Amanda V., Barichello, Tatiana, Quevedo, João, Dal-Pizzol, Felipe, Gelain, Daniel P.
Médium: Artigo
Jazyk:Inglês
Vydáno: American Society for Biochemistry and Molecular Biology 2018
Témata:
Molecular Bases of Disease
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5766916/
https://ncbi.nlm.nih.gov/pubmed/29127203
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M117.786756
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