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Modelling the effects of chloroquine on KCNJ2-linked short QT syndrome
A gain-of-function KCNJ2 D172N mutation in KCNJ2-encoded Kir2.1 channels underlies one form of short QT syndrome (SQT3), which is associated with increased susceptibility to arrhythmias and sudden death. Anti-malarial drug chloroquine was reported as an effective inhibitor of Kir2.1 channels. Using...
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| Yayımlandı: | Oncotarget |
|---|---|
| Asıl Yazarlar: | , , |
| Materyal Türü: | Artigo |
| Dil: | Inglês |
| Baskı/Yayın Bilgisi: |
Impact Journals LLC
2017
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| Konular: | |
| Online Erişim: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5739752/ https://ncbi.nlm.nih.gov/pubmed/29290967 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.22490 |
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