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A computational model for regulation of nanoscale glucan exposure in Candida albicans
Candida albicans is a virulent human opportunistic pathogen. It evades innate immune surveillance by masking an immunogenic cell wall polysaccharide, β-glucan, from recognition by the immunoreceptor Dectin-1. Glucan unmasking by the antifungal drug caspofungin leads to changes in the nanostructure o...
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| I publikationen: | PLoS One |
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| Huvudupphovsmän: | , , |
| Materialtyp: | Artigo |
| Språk: | Inglês |
| Publicerad: |
Public Library of Science
2017
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| Ämnen: | |
| Länkar: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5726713/ https://ncbi.nlm.nih.gov/pubmed/29232689 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0188599 |
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