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HIV-1 counteracts an innate restriction by amyloid precursor protein resulting in neurodegeneration
While beta-amyloid (Aβ), a classic hallmark of Alzheimer’s disease (AD) and dementia, has long been known to be elevated in the human immunodeficiency virus type 1 (HIV-1)-infected brain, why and how Aβ is produced, along with its contribution to HIV-associated neurocognitive disorder (HAND) remains...
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| Veröffentlicht in: | Nat Commun |
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| Hauptverfasser: | , , , , , , |
| Format: | Artigo |
| Sprache: | Inglês |
| Veröffentlicht: |
Nature Publishing Group UK
2017
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| Schlagworte: | |
| Online Zugang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5688069/ https://ncbi.nlm.nih.gov/pubmed/29142315 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/s41467-017-01795-8 |
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