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Intrahippocampal Administration of Amyloid-β(1–42) Oligomers Acutely Impairs Spatial Working Memory, Insulin Signaling, and Hippocampal Metabolism
Increasing evidence suggests that abnormal brain accumulation of amyloid-β(1–42) (Aβ(1–42)) oligomers plays a causal role in Alzheimer’s disease (AD), and in particular may cause the cognitive deficits that are the hallmark of AD. In vitro, Aβ(1–42) oligomers impair insulin signaling and suppress ne...
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| Publicado no: | J Alzheimers Dis |
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| Main Authors: | , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2012
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5674792/ https://ncbi.nlm.nih.gov/pubmed/22430529 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3233/JAD-2012-112192 |
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