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Intrahippocampal Administration of Amyloid-β(1–42) Oligomers Acutely Impairs Spatial Working Memory, Insulin Signaling, and Hippocampal Metabolism
Increasing evidence suggests that abnormal brain accumulation of amyloid-β(1–42) (Aβ(1–42)) oligomers plays a causal role in Alzheimer’s disease (AD), and in particular may cause the cognitive deficits that are the hallmark of AD. In vitro, Aβ(1–42) oligomers impair insulin signaling and suppress ne...
Gorde:
| Argitaratua izan da: | J Alzheimers Dis |
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| Egile Nagusiak: | , |
| Formatua: | Artigo |
| Hizkuntza: | Inglês |
| Argitaratua: |
2012
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| Gaiak: | |
| Sarrera elektronikoa: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5674792/ https://ncbi.nlm.nih.gov/pubmed/22430529 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3233/JAD-2012-112192 |
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