A protein scaffold coordinates SRC-mediated JNK activation in response to metabolic stress

Obesity is a major risk factor for the development of metabolic syndrome and type 2 diabetes. How obesity contributes to metabolic syndrome is unclear. Free fatty acid (FFA) activation of a non-receptor tyrosine kinase (SRC)-dependent cJun NH(2)-terminal kinase (JNK) signaling pathway is implicated...

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Publicat a:Cell Rep
Autors principals: Kant, Shashi, Standen, Claire L., Morel, Caroline, Jung, Dae Young, Kim, Jason K., Swat, Wojciech, Flavell, Richard A., Davis, Roger J.
Format: Artigo
Idioma:Inglês
Publicat: 2017
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Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC5659285/
https://ncbi.nlm.nih.gov/pubmed/28930674
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.celrep.2017.08.025
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