Nicotine suppresses apoptosis by regulating α7nAChR/Prx1 axis in oral precancerous lesions

Nicotine, a tumor promoter in tobacco, can increase Peroxiredoxin (Prx1) and nicotinic acetylcholine receptors (nAChRs) in oral squamous cell carcinoma (OSCC). In the present study, we investigate the effects of nicotine in oral precancerous lesions focusing on apoptosis and nAChR/Prx1 signaling. We...

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Bibliographic Details
Published in:	Oncotarget
Main Authors:	Wang, Chunxiao, Niu, Wenwen, Chen, Hui, Shi, Ni, He, Dian, Zhang, Min, Ge, Lihua, Tian, Zhenchuan, Qi, Moci, Chen, Tong, Tang, Xiaofei
Format:	Artigo
Language:	Inglês
Published:	Impact Journals LLC 2017
Subjects:	Research Paper
Online Access:	https://ncbi.nlm.nih.gov/pmc/articles/PMC5650400/ https://ncbi.nlm.nih.gov/pubmed/29088845 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.20506
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Nicotine suppresses apoptosis by regulating α7nAChR/Prx1 axis in oral precancerous lesions

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