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Prenatal nicotinic exposure prolongs superior laryngeal C-fiber–mediated apnea and bradycardia through enhancing neuronal TRPV1 expression and excitation

Maternal cigarette smoke, including prenatal nicotinic exposure (PNE), is responsible for sudden infant death syndrome (SIDS). The fatal events of SIDS are characterized by severe bradycardia and life-threatening apneas. Although activation of transient receptor potential vanilloid 1 (TRPV1) of supe...

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Bibliografiske detaljer
Udgivet i:FASEB J
Main Authors: Gao, Xiuping, Zhao, Lei, Zhuang, Jianguo, Zang, Na, Xu, Fadi
Format: Artigo
Sprog:Inglês
Udgivet: Federation of American Societies for Experimental Biology 2017
Fag:
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC5602895/
https://ncbi.nlm.nih.gov/pubmed/28615326
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.201700163R
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