Prenatal nicotinic exposure prolongs superior laryngeal C-fiber–mediated apnea and bradycardia through enhancing neuronal TRPV1 expression and excitation

Maternal cigarette smoke, including prenatal nicotinic exposure (PNE), is responsible for sudden infant death syndrome (SIDS). The fatal events of SIDS are characterized by severe bradycardia and life-threatening apneas. Although activation of transient receptor potential vanilloid 1 (TRPV1) of supe...

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Bibliographic Details
Published in:FASEB J
Main Authors: Gao, Xiuping, Zhao, Lei, Zhuang, Jianguo, Zang, Na, Xu, Fadi
Format: Artigo
Language:Inglês
Published: Federation of American Societies for Experimental Biology 2017
Subjects:
Research
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC5602895/
https://ncbi.nlm.nih.gov/pubmed/28615326
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.201700163R
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