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Prenatal nicotinic exposure prolongs superior laryngeal C-fiber–mediated apnea and bradycardia through enhancing neuronal TRPV1 expression and excitation
Maternal cigarette smoke, including prenatal nicotinic exposure (PNE), is responsible for sudden infant death syndrome (SIDS). The fatal events of SIDS are characterized by severe bradycardia and life-threatening apneas. Although activation of transient receptor potential vanilloid 1 (TRPV1) of supe...
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| Udgivet i: | FASEB J |
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| Main Authors: | , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
Federation of American Societies for Experimental Biology
2017
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5602895/ https://ncbi.nlm.nih.gov/pubmed/28615326 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.201700163R |
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