Reduced FAK-STAT3 signaling contributes to ER stress-induced mitochondrial dysfunction and death in endothelial cells

Excessive endoplasmic reticulum (ER) stress leads to cell loss in many diseases, e.g., contributing to endothelial cell loss after spinal cord injury. Here, we determined whether ER stress-induced mitochondrial dysfunction could be explained by interruption of the focal adhesion kinase (FAK)-mitocho...

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Publicado en:Cell Signal
Autores principales: Banerjee, Kalpita, Keasey, Matt P., Razskazovskiy, Vladislav, Visavadiya, Nishant P., Jia, Cuihong, Hagg, Theo
Formato: Artigo
Lenguaje:Inglês
Publicado: 2017
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Article
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC5589129/
https://ncbi.nlm.nih.gov/pubmed/28495589
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cellsig.2017.05.007
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