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Histone H3 N-terminal mutations allow hyperactivation of the yeast GAL1 gene in vivo.
Recent work has shown that the yeast histone H4 N-terminus, while not essential for viability, is required for repression of the silent mating loci and activation of GAL1 and PHO5 promoters. Because histone H3 shares many structural features with histone H4 and is intimately associated with H4 in th...
Bewaard in:
| Gepubliceerd in: | EMBO J |
|---|---|
| Hoofdauteurs: | , |
| Formaat: | Artigo |
| Taal: | Inglês |
| Gepubliceerd in: |
Nature Publishing Group
1992
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| Onderwerpen: | |
| Online toegang: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC556864/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/1505519/ https://ncbi.nlm.nih.govhttps://doi.org/10.1002/j.1460-2075.1992.tb05408.x |
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