Yeast histone H3 and H4 N termini function through different GAL1 regulatory elements to repress and activate transcription.
Previous work has shown that N-terminal deletions of yeast histone H3 cause a 2- to 4-fold increase in the induction of GAL1 and a number of other genes involved in galactose metabolism. In contrast, deletions at the H4 N terminus cause a 10- to 20-fold decrease in the induction of these same GAL ge...
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| Publicat a: | Proc Natl Acad Sci U S A |
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| Autors principals: | , , |
| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
National Academy of Sciences
1995
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC41757/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/7777566/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.92.12.5664 |
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