Oral apolipoprotein A-I mimetic peptide improves cognitive function and reduces amyloid burden in a mouse model of Alzheimer’s disease

Recent evidence indicates that inflammation may significantly contribute to the pathogenesis of Alzheimer’s disease (AD). Since the apo A-I mimetic peptide D-4F has been shown to inhibit atherosclerotic lesion formation and regress already existing lesions (in the presence of pravastatin) and the pe...

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Dades bibliogràfiques
Publicat a:Neurobiol Dis
Autors principals: Handattu, Shaila P., Garber, David W., Monroe, Candyce E., van Groen, Thomas, Kadish, Inga, Nayyar, Gaurav, Cao, Dongfeng, Palgunachari, Mayakonda N., Li, Ling, Anantharamaiah, G.M.
Format: Artigo
Idioma:Inglês
Publicat: 2009
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Article
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC5558244/
https://ncbi.nlm.nih.gov/pubmed/19344763
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.nbd.2009.03.007
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