טוען...
Selective glucocorticoid receptor modulators (SGRMs) delay castrate-resistant prostate cancer growth
Increased glucocorticoid receptor (GR) expression and activity following androgen blockade can contribute to castration-resistant prostate cancer (CRPC) progression. Therefore, we hypothesized that GR antagonism will have therapeutic benefit in CRPC. However, the FDA-approved nonselective, steroidal...
שמור ב:
| הוצא לאור ב: | Mol Cancer Ther |
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| Main Authors: | , , , , , , , , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2017
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5544558/ https://ncbi.nlm.nih.gov/pubmed/28428441 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1158/1535-7163.MCT-16-0923 |
| תגים: |
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