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Thiamet G mediates neuroprotection in experimental stroke by modulating microglia/macrophage polarization and inhibiting NF-κB p65 signaling

Inflammatory responses are accountable for secondary injury induced by acute ischemic stroke (AIS). Previous studies indicated that O-GlcNAc modification (O-GlcNAcylation) is involved in the pathology of AIS, and increase of O-GlcNAcylation by glucosamine attenuated the brain damage after ischemia/r...

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Detalhes bibliográficos
Publicado no:J Cereb Blood Flow Metab
Main Authors: He, Yating, Ma, Xiaofeng, Li, Daojing, Hao, Junwei
Formato: Artigo
Idioma:Inglês
Publicado em: SAGE Publications 2016
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5536801/
https://ncbi.nlm.nih.gov/pubmed/27864466
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/0271678X16679671
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