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Dysfunctional GPR40/FFAR1 signaling exacerbates pain behavior in mice
We previously showed that activation of G protein-coupled receptor 40/free fatty acid receptor 1 (GPR40/FFAR1) signaling modulates descending inhibition of pain. In this study, we investigated the involvement of fatty acid-GPR40/FFAR1 signaling in the transition from acute to chronic pain. We used G...
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| 發表在: | PLoS One |
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| Main Authors: | , , , , , , , , , , |
| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
Public Library of Science
2017
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5516985/ https://ncbi.nlm.nih.gov/pubmed/28723961 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0180610 |
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