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Loss of WDFY3 Ameliorates Severity of Serum Transfer-Induced Arthritis Independently of Autophagy
WDFY3 is a master regulator of selective autophagy that we recently showed to interact with TRAF6 and augment RANKL-induced osteoclastogenesis in vitro and in vivo via the NF-κB pathway. Since the NF-κB pathway plays a major role in inflammation herein, we investigate the role of WDFY3 in an arthrit...
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| Publicado no: | Cell Immunol |
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| Main Authors: | , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2017
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5515728/ https://ncbi.nlm.nih.gov/pubmed/28449847 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cellimm.2017.04.001 |
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