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Elevated systemic TGF-β impairs aortic vasomotor function through activation of NADPH oxidase-driven superoxide production and leads to hypertension, myocardial remodeling, and increased plaque formation in apoE(−/−) mice

The role of circulating, systemic TGF-β levels in endothelial function is not clear. TGF-β(1) may cause endothelial dysfunction in apolipoprotein E-deficient (apoE(−/−)) mice via stimulation of reactive oxygen species (ROS) production by the NADPH oxidase (NOX) system and aggravate aortic and heart...

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書目詳細資料
發表在:	Am J Physiol Heart Circ Physiol
Main Authors:	Buday, Anna, Őrsy, Petra, Godó, Mária, Mózes, Miklós, Kökény, Gábor, Lacza, Zsombor, Koller, Ákos, Ungvári, Zoltán, Gross, Marie-Luise, Benyó, Zoltán, Hamar, Péter
格式:	Artigo
語言:	Inglês
出版:	American Physiological Society 2010
主題:	Articles
在線閱讀:	https://ncbi.nlm.nih.gov/pmc/articles/PMC5504387/ https://ncbi.nlm.nih.gov/pubmed/20511416 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajpheart.01042.2009
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Elevated systemic TGF-β impairs aortic vasomotor function through activation of NADPH oxidase-driven superoxide production and leads to hypertension, myocardial remodeling, and increased plaque formation in apoE(−/−) mice

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