Deregulation of mitochondrial F1FO-ATP synthase via OSCP in Alzheimer’s disease

F1FO-ATP synthase is critical for mitochondrial functions. The deregulation of this enzyme results in dampened mitochondrial oxidative phosphorylation (OXPHOS) and activated mitochondrial permeability transition (mPT), defects which accompany Alzheimer’s disease (AD). However, the molecular mechanis...

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Pubblicato in:Nat Commun
Autori principali: Beck, Simon J., Guo, Lan, Phensy, Aarron, Tian, Jing, Wang, Lu, Tandon, Neha, Gauba, Esha, Lu, Lin, Pascual, Juan M., Kroener, Sven, Du, Heng
Natura: Artigo
Lingua:Inglês
Pubblicazione: Nature Publishing Group 2016
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Article
Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC5494197/
https://ncbi.nlm.nih.gov/pubmed/27151236
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/ncomms11483
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