Deregulation of mitochondrial F1FO-ATP synthase via OSCP in Alzheimer’s disease

F1FO-ATP synthase is critical for mitochondrial functions. The deregulation of this enzyme results in dampened mitochondrial oxidative phosphorylation (OXPHOS) and activated mitochondrial permeability transition (mPT), defects which accompany Alzheimer’s disease (AD). However, the molecular mechanis...

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Bibliografski detalji
Izdano u:Nat Commun
Glavni autori: Beck, Simon J., Guo, Lan, Phensy, Aarron, Tian, Jing, Wang, Lu, Tandon, Neha, Gauba, Esha, Lu, Lin, Pascual, Juan M., Kroener, Sven, Du, Heng
Format: Artigo
Jezik:Inglês
Izdano: Nature Publishing Group 2016
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Article
Online pristup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5494197/
https://ncbi.nlm.nih.gov/pubmed/27151236
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/ncomms11483
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