Roles of Tumor Necrosis Factor Alpha (TNF-α) and the p55 TNF Receptor in CD1d Induction and Coxsackievirus B3-Induced Myocarditis

Podobne zapisy

• Tumor Necrosis Factor-α Promotes Myocarditis in Female Mice Infected with Coxsackievirus B3 Through Upregulation of CD1d on Hematopoietic Cells
  od: Huber, Sally
  Wydane: (2010)
• Vγ4(+) T Cells Promote Autoimmune CD8(+) Cytolytic T-Lymphocyte Activation in Coxsackievirus B3-Induced Myocarditis in Mice: Role for CD4(+) Th1 Cells
  od: Huber, S. A., i wsp.
  Wydane: (2002)
• Human Cytomegalovirus Infection Inhibits Tumor Necrosis Factor Alpha (TNF-α) Signaling by Targeting the 55-Kilodalton TNF-α Receptor
  od: Baillie, J., i wsp.
  Wydane: (2003)
• Elevated Levels of Tumor Necrosis Factor Alpha (TNF-α) in Human Immunodeficiency Virus Type 1-Transgenic Mice: Prevention of Death by Antibody to TNF-α
  od: De, Swapan K., i wsp.
  Wydane: (2002)
• Hormonal Regulation of CD4(+) T-Cell Responses in Coxsackievirus B3-Induced Myocarditis in Mice
  od: Huber, S. A., i wsp.
  Wydane: (1999)