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Endogenous nitric oxide formation in cardiac myocytes does not control respiration during β‐adrenergic stimulation
KEY POINTS: In the heart, endothelial nitric oxide (NO) controls oxygen consumption in the working heart through paracrine mechanisms. While cardiac myocytes contain several isoforms of NO synthases, it is unclear whether these can control respiration in an intracrine fashion. A long‐standing contro...
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| Publicado no: | J Physiol |
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| Principais autores: | , , , , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
John Wiley and Sons Inc.
2017
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5471411/ https://ncbi.nlm.nih.gov/pubmed/28229450 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1113/JP273750 |
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