Infection of vascular endothelial cells with herpes simplex virus enhances tissue factor activity and reduces thrombomodulin expression.

Latent infection of vascular cells with herpes-viruses may play a pathogenic role in the development of human atherosclerosis. In a previous study, we found that cultured human umbilical vein endothelial cells (HUVECs) infected with herpes simplex virus 1 (HSV-1) became procoagulant, exemplified bot...

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Bibliografiske detaljer
Main Authors: Key, N S, Vercellotti, G M, Winkelmann, J C, Moldow, C F, Goodman, J L, Esmon, N L, Esmon, C T, Jacob, H S
Format: Artigo
Sprog:Inglês
Udgivet: 1990
Fag:
Research Article
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC54690/
https://ncbi.nlm.nih.gov/pubmed/2169619
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