Infection of vascular endothelial cells with herpes simplex virus enhances tissue factor activity and reduces thrombomodulin expression.

Latent infection of vascular cells with herpes-viruses may play a pathogenic role in the development of human atherosclerosis. In a previous study, we found that cultured human umbilical vein endothelial cells (HUVECs) infected with herpes simplex virus 1 (HSV-1) became procoagulant, exemplified bot...

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Detalles Bibliográficos

Publicado en:	Proc Natl Acad Sci U S A
Principais autores:	Key, N S, Vercellotti, G M, Winkelmann, J C, Moldow, C F, Goodman, J L, Esmon, N L, Esmon, C T, Jacob, H S
Formato:	Artigo
Idioma:	Inglês
Publicado:	National Academy of Sciences 1990
Assuntos:	Research Article
Acceso en liña:	https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC54690/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/2169619/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.87.18.7095
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