DNA octamer element can confer E1A trans-activation, and adenovirus infection results in a stimulation of the DNA-binding activity of OTF-1/NFIII factor.

Podobne knjige/članki

• Anti-OTF-1 antibodies inhibit NFIII stimulation of in vitro adenovirus DNA replication.
  od: Pruijn, J M, et al.
  Izdano: (1989)
• Activation of octamer-containing promoters by either octamer-binding transcription factor 1 (OTF-1) or OTF-2 and requirement of an additional B-cell-specific component for optimal transcription of immunoglobulin promoters.
  od: Pierani, A, et al.
  Izdano: (1990)
• Transcription factors NFI and NFIII/oct-1 function independently, employing different mechanisms to enhance adenovirus DNA replication.
  od: Mul, Y M, et al.
  Izdano: (1990)
• The NFIII/OCT-1 binding site stimulates adenovirus DNA replication in vivo and is functionally redundant with adjacent sequences.
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  Izdano: (1993)
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  od: Broggini, M, et al.
  Izdano: (1989)