DNA octamer element can confer E1A trans-activation, and adenovirus infection results in a stimulation of the DNA-binding activity of OTF-1/NFIII factor.

Lignende værker

• Anti-OTF-1 antibodies inhibit NFIII stimulation of in vitro adenovirus DNA replication.
  af: Pruijn, J M, et al.
  Udgivet: (1989)
• Activation of octamer-containing promoters by either octamer-binding transcription factor 1 (OTF-1) or OTF-2 and requirement of an additional B-cell-specific component for optimal transcription of immunoglobulin promoters.
  af: Pierani, A, et al.
  Udgivet: (1990)
• Transcription factors NFI and NFIII/oct-1 function independently, employing different mechanisms to enhance adenovirus DNA replication.
  af: Mul, Y M, et al.
  Udgivet: (1990)
• The NFIII/OCT-1 binding site stimulates adenovirus DNA replication in vivo and is functionally redundant with adjacent sequences.
  af: Hatfield, L, et al.
  Udgivet: (1993)
• Distamycins inhibit the binding of OTF-1 and NFE-1 transfactors to their conserved DNA elements.
  af: Broggini, M, et al.
  Udgivet: (1989)