FGF23 Is Not Required to Regulate Fetal Phosphorus Metabolism but Exerts Effects Within 12 Hours After Birth

Loss of fibroblast growth factor-23 (FGF23) causes hyperphosphatemia, extraskeletal calcifications, and early mortality; excess FGF23 causes hypophosphatemia with rickets or osteomalacia. However, FGF23 may not be important during fetal development. FGF23 deficiency (Fgf23 null) and FGF23 excess (Ph...

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Dades bibliogràfiques
Publicat a:Endocrinology
Autors principals: Ma, Yue, Kirby, Beth J., Fairbridge, Nicholas A., Karaplis, Andrew C., Lanske, Beate, Kovacs, Christopher S.
Format: Artigo
Idioma:Inglês
Publicat: Endocrine Society 2016
Matèries:
Research Articles
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC5413075/
https://ncbi.nlm.nih.gov/pubmed/27929669
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1210/en.2016-1369
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