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Amelioration of amyloid-β-induced deficits by DcR3 in an Alzheimer’s disease model
BACKGROUND: Microglia mediate amyloid-beta peptide (Aβ)-induced neuroinflammation, which is one of the key events in the pathogenesis of Alzheimer’s disease (AD). Decoy receptor 3 (DcR3)/TNFRSF6B is a pleiotropic immunomodulator that promotes macrophage differentiation toward the M2 anti-inflammator...
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| Udgivet i: | Mol Neurodegener |
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| Main Authors: | , , , , , |
| Format: | Artigo |
| Sprog: | Inglês |
| Udgivet: |
BioMed Central
2017
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| Fag: | |
| Online adgang: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5402663/ https://ncbi.nlm.nih.gov/pubmed/28438208 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/s13024-017-0173-0 |
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