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Interference with Ca(2+)-Dependent Proteolysis Does Not Alter the Course of Muscle Wasting in Experimental Cancer Cachexia

Protein hypercatabolism significantly contributes to the onset and progression of muscle wasting in cancer cachexia. In this regard, a major role is played by the ATP-ubiquitin-proteasome-dependent pathway and by autophagy. However, little is known about the relevance of the Ca(2+)-dependent proteol...

תיאור מלא

שמור ב:
מידע ביבליוגרפי
הוצא לאור ב:Front Physiol
Main Authors: Pin, Fabrizio, Minero, Valerio G., Penna, Fabio, Muscaritoli, Maurizio, De Tullio, Roberta, Baccino, Francesco M., Costelli, Paola
פורמט: Artigo
שפה:Inglês
יצא לאור: Frontiers Media S.A. 2017
נושאים:
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC5395607/
https://ncbi.nlm.nih.gov/pubmed/28469577
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.3389/fphys.2017.00213
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