RelA/p65 inhibition prevents tendon adhesion by modulating inflammation, cell proliferation, and apoptosis

Peritendinous tissue fibrosis which leads to poor tendon function is a worldwide clinical problem; however, its mechanism remains unclear. Transcription factor RelA/p65, an important subunit in the NF-κB complex, is known to have a critical role in many fibrotic diseases. Here, we show that RelA/p65...

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Bibliografiska uppgifter
I publikationen:Cell Death Dis
Huvudupphovsmän: Chen, Shuai, Jiang, Shichao, Zheng, Wei, Tu, Bing, Liu, Shen, Ruan, Hongjiang, Fan, Cunyi
Materialtyp: Artigo
Språk:Inglês
Publicerad: Nature Publishing Group 2017
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Original Article
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC5386538/
https://ncbi.nlm.nih.gov/pubmed/28358376
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/cddis.2017.135
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