Deficiency of cardiomyocyte-specific microRNA-378 contributes to the development of cardiac fibrosis involving a transforming growth factor β (TGFβ1)-dependent paracrine mechanism.

Podobne knjige/članki

• Deficiency of Cardiomyocyte-specific MicroRNA-378 Contributes to the Development of Cardiac Fibrosis Involving a Transforming Growth Factor β (TGFβ1)-dependent Paracrine Mechanism
  od: Nagalingam, Raghu S., et al.
  Izdano: (2014)
• A cardiac-enriched microRNA, miR-378, blocks cardiac hypertrophy by targeting Ras signaling.
  od: Nagalingam, Raghu S., et al.
  Izdano: (2017)
• A Novel Cardiomyocyte-enriched MicroRNA, miR-378, Targets Insulin-like Growth Factor 1 Receptor: IMPLICATIONS IN POSTNATAL CARDIAC REMODELING AND CELL SURVIVAL
  od: Knezevic, Ivana, et al.
  Izdano: (2012)
• A Cardiac-enriched MicroRNA, miR-378, Blocks Cardiac Hypertrophy by Targeting Ras Signaling
  od: Nagalingam, Raghu S., et al.
  Izdano: (2013)
• SIRT3 Blocks Aging-Associated Tissue Fibrosis in Mice by Deacetylating and Activating Glycogen Synthase Kinase 3β
  od: Sundaresan, Nagalingam R., et al.
  Izdano: (2016)