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DNA repair interacts with autophagy to regulate inflammatory responses to pulmonary hyperoxia
Oxygen is supplied as supportive treatment for patients suffering from acute respiratory distress syndrome. Unfortunately, high oxygen concentration increases reactive oxygen species (ROS) generation, which causes DNA damage and ultimately cell death in the lung. Although 8-oxoguanine-DNA glycosylas...
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| 發表在: | J Immunol |
|---|---|
| Main Authors: | , , , , , , , , , , , , |
| 格式: | Artigo |
| 語言: | Inglês |
| 出版: |
2017
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| 主題: | |
| 在線閱讀: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5360514/ https://ncbi.nlm.nih.gov/pubmed/28202616 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1601001 |
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