DNA repair interacts with autophagy to regulate inflammatory responses to pulmonary hyperoxia

Oxygen is supplied as supportive treatment for patients suffering from acute respiratory distress syndrome. Unfortunately, high oxygen concentration increases reactive oxygen species (ROS) generation, which causes DNA damage and ultimately cell death in the lung. Although 8-oxoguanine-DNA glycosylas...

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Detalhes bibliográficos
Publicado no:J Immunol
Main Authors: Ye, Yan, Lin, Ping, Zhang, Weidong, Tan, Shirui, Zhou, Xikun, Li, Rongpeng, Pu, Qinqin, Koff, Jonathan, Dhasarathy, Archana, Ma, Feng, Deng, Xin, Jiang, Jianxin, Wu, Min
Formato: Artigo
Idioma:Inglês
Publicado em: 2017
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC5360514/
https://ncbi.nlm.nih.gov/pubmed/28202616
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1601001
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