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Signaling by FGF Receptor 2, Not FGF Receptor 1, Regulates Myelin Thickness through Activation of ERK1/2–MAPK, Which Promotes mTORC1 Activity in an Akt-Independent Manner

FGF signaling has emerged as a significant “late-stage” regulator of myelin thickness in the CNS, independent of oligodendrocyte differentiation. Therefore, it is critically important to identify the specific FGF receptor type and its downstream signaling molecules in oligodendrocytes to obtain bett...

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Bibliografiska uppgifter
I publikationen:J Neurosci
Huvudupphovsmän: Furusho, Miki, Ishii, Akihiro, Bansal, Rashmi
Materialtyp: Artigo
Språk:Inglês
Publicerad: Society for Neuroscience 2017
Ämnen:
Länkar:https://ncbi.nlm.nih.gov/pmc/articles/PMC5354334/
https://ncbi.nlm.nih.gov/pubmed/28193689
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1523/JNEUROSCI.3316-16.2017
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