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Endostatin inhibits androgen-independent prostate cancer growth by suppressing nuclear receptor-mediated oxidative stress
Androgen-deprivation therapy has been identified to induce oxidative stress in prostate cancer (PCa), leading to reactivation of androgen receptor (AR) signaling in a hormone-refractory manner. Thus, antioxidant therapies have gained attention as adjuvants for castration-resistant PCa. Here, we repo...
Αποθηκεύτηκε σε:
| Τόπος έκδοσης: | FASEB J |
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| Κύριοι συγγραφείς: | , , , , , , , , |
| Μορφή: | Artigo |
| Γλώσσα: | Inglês |
| Έκδοση: |
Federation of American Societies for Experimental Biology
2017
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| Θέματα: | |
| Διαθέσιμο Online: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5349799/ https://ncbi.nlm.nih.gov/pubmed/28069826 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.201601178R |
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