Ginsenoside Rb1 Inhibits Doxorubicin-Triggered H9C2 Cell Apoptosis via Aryl Hydrocarbon Receptor

Doxorubicin (DOX) is a highly effective chemotherapeutic agent; however, the dose-dependent cardiotoxicity associated with DOX significantly limits its clinical application. In the present study, we investigated whether Rb1 could prevent DOX-induced apoptosis in H9C2 cells via aryl hydrocarbon recep...

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Bibliografske podrobnosti
izdano v:Biomol Ther (Seoul)
Main Authors: Zhang, Yaxin, Wang, Yuguang, Ma, Zengchun, Liang, Qiande, Tang, Xianglin, Tan, Hongling, Xiao, Chengrong, Gao, Yue
Format: Artigo
Jezik:Inglês
Izdano: The Korean Society of Applied Pharmacology 2017
Teme:
Original Article
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC5340546/
https://ncbi.nlm.nih.gov/pubmed/27829271
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4062/biomolther.2016.066
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