Response to ‘Refined analyses suggest that recombination is a minor source of genomic diversity in Pseudomonas aeruginosa chronic cystic fibrosis infections’ by Williams et al. (2016)

Míreanna Comhchosúla

• Recombination is a key driver of genomic and phenotypic diversity in a Pseudomonas aeruginosa population during cystic fibrosis infection
  le: Darch, Sophie E., et al.
  Foilsithe: (2015)
• Refined analyses suggest that recombination is a minor source of genomic diversity in Pseudomonas aeruginosa chronic cystic fibrosis infections
  le: Williams, David, et al.
  Foilsithe: (2016)
• Competition in Biofilms between Cystic Fibrosis Isolates of Pseudomonas aeruginosa Is Shaped by R-Pyocins
  le: Oluyombo, Olubukola, et al.
  Foilsithe: (2019)
• Compensatory evolution of Pseudomonas aeruginosa’s slow growth phenotype suggests mechanisms of adaptation in cystic fibrosis
  le: La Rosa, Ruggero, et al.
  Foilsithe: (2021)
• Heterogenous Susceptibility to R-Pyocins in Populations of Pseudomonas aeruginosa Sourced from Cystic Fibrosis Lungs
  le: Mei, Madeline, et al.
  Foilsithe: (2021)