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Revealing the Concealed Nature of Long QT Type 3 Syndrome
BACKGROUND: Gain-of-function mutations in the voltage-gated sodium channel (Nav1.5) are associated with the long QT-3 (LQT3) syndrome. Nav1.5 is densely expressed at the intercalated disk, and narrow intercellular separation can modulate cell-to-cell coupling via extracellular electric fields and de...
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| 出版年: | Circ Arrhythm Electrophysiol |
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| 主要な著者: | , , , |
| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2017
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5319726/ https://ncbi.nlm.nih.gov/pubmed/28213505 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/CIRCEP.116.004400 |
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