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Hypoxia upregulates HIG2 expression and contributes to bevacizumab resistance in glioblastoma
Hypoxia contributes to the maintenance of stem-like cells in glioblastoma (GBM), and activates vascular mimicry and tumor resistance to anti-angiogenesis treatments. The present study examined the expression patterns and biological significance of hypoxia-inducible protein 2 (HIG2, also known as HIL...
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| Vydáno v: | Oncotarget |
|---|---|
| Hlavní autoři: | , , , , , , , , , , , , , , , |
| Médium: | Artigo |
| Jazyk: | Inglês |
| Vydáno: |
Impact Journals LLC
2016
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| Témata: | |
| On-line přístup: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5216980/ https://ncbi.nlm.nih.gov/pubmed/27329597 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.18632/oncotarget.10029 |
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