Expression of the human apolipoprotein A-I gene in transgenic mice alters high density lipoprotein (HDL) particle size distribution and diminishes selective uptake of HDL cholesteryl esters.

Antzeko izenburuak

• Hypertriglyceridemia and cholesteryl ester transfer protein interact to dramatically alter high density lipoprotein levels, particle sizes, and metabolism. Studies in transgenic mice.
  nork: Hayek, T, et al.
  Argitaratua: (1993)
• An interaction between the human cholesteryl ester transfer protein (CETP) and apolipoprotein A-I genes in transgenic mice results in a profound CETP-mediated depression of high density lipoprotein cholesterol levels.
  nork: Hayek, T, et al.
  Argitaratua: (1992)
• Mechanism of hypertriglyceridemia in human apolipoprotein (apo) CIII transgenic mice. Diminished very low density lipoprotein fractional catabolic rate associated with increased apo CIII and reduced apo E on the particles.
  nork: Aalto-Setälä, K, et al.
  Argitaratua: (1992)
• Decreased selective uptake of high density lipoprotein cholesteryl esters in apolipoprotein E knock-out mice
  nork: Arai, Takeshi, et al.
  Argitaratua: (1999)
• Scavenger receptor B1 (SR-B1) profoundly excludes high density lipoprotein (HDL) apolipoprotein AII as it nibbles HDL-cholesteryl ester
  nork: Gillard, Baiba K., et al.
  Argitaratua: (2017)