Heme oxygenase-2 gene deletion attenuates oxidative stress in neurons exposed to extracellular hemin
BACKGROUND: Hemin, the oxidized form of heme, accumulates in intracranial hematomas and is a potent oxidant. Growing evidence suggests that it contributes to delayed injury to surrounding tissue, and that this process is affected by the heme oxygenase enzymes. In a prior study, heme oxygenase-2 gene...
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| Publicado en: | BMC Neurosci |
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| Principais autores: | , , |
| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado: |
BMC
2004
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| Assuntos: | |
| Acceso en liña: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC521072/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/15377391/ https://ncbi.nlm.nih.govhttps://doi.org/10.1186/1471-2202-5-34 |
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