The tumor suppressor FLCN mediates an alternate mTOR pathway to regulate browning of adipose tissue

Noncanonical mechanistic target of rapamycin (mTOR) pathways remain poorly understood. Mutations in the tumor suppressor folliculin (FLCN) cause Birt-Hogg-Dubé syndrome, a hamartomatous disease marked by mitochondria-rich kidney tumors. FLCN functionally interacts with mTOR and is expressed in most...

詳細記述

保存先:
書誌詳細
出版年:Genes Dev
主要な著者: Wada, Shogo, Neinast, Michael, Jang, Cholsoon, Ibrahim, Yasir H., Lee, Gina, Babu, Apoorva, Li, Jian, Hoshino, Atsushi, Rowe, Glenn C., Rhee, James, Martina, José A., Puertollano, Rosa, Blenis, John, Morley, Michael, Baur, Joseph A., Seale, Patrick, Arany, Zoltan
フォーマット: Artigo
言語:Inglês
出版事項: Cold Spring Harbor Laboratory Press 2016
主題:
Research Paper
オンライン･アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC5159669/
https://ncbi.nlm.nih.gov/pubmed/27913603
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1101/gad.287953.116
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