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Toll-interacting protein deficiency promotes neurodegeneration via impeding autophagy completion in high-fat diet-fed ApoE(−/−) mouse model
The excessive accumulation of specific cellular proteins or autophagic vacuoles (AVs) within neurons is a pathologic hallmark of neurodegenerative diseases. Constitutive autophagy in neurons prevents abnormal intracellular protein aggregation and is critical for maintaining cell survival. Since our...
שמור ב:
| הוצא לאור ב: | Brain Behav Immun |
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| Main Authors: | , , , , , , , |
| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
2016
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5154796/ https://ncbi.nlm.nih.gov/pubmed/27720815 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.bbi.2016.10.002 |
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