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Purinergic signaling triggers endfoot high-amplitude Ca(2+) signals and causes inversion of neurovascular coupling after subarachnoid hemorrhage
Neurovascular coupling supports brain metabolism by matching focal increases in neuronal activity with local arteriolar dilation. Previously, we demonstrated that an emergence of spontaneous endfoot high-amplitude Ca(2+) signals (eHACSs) caused a pathologic shift in neurovascular coupling from vasod...
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| Publicado en: | J Cereb Blood Flow Metab |
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| Autores principales: | , , |
| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
SAGE Publications
2016
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC5094310/ https://ncbi.nlm.nih.gov/pubmed/27207166 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1177/0271678X16650911 |
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