Systemic anaphylaxis in the mouse can be mediated largely through IgG1 and Fc gammaRIII. Assessment of the cardiopulmonary changes, mast cell degranulation, and death associated with active or IgE- or IgG1-dependent passive anaphylaxis.

Documenti analoghi

• Absence of Fc epsilonRI alpha chain results in upregulation of Fc gammaRIII-dependent mast cell degranulation and anaphylaxis. Evidence of competition between Fc epsilonRI and Fc gammaRIII for limiting amounts of FcR beta and gamma chains.
  di: Dombrowicz, D, et al.
  Pubblicazione: (1997)
• Anti-FcγRIIB mAb suppresses murine IgG-dependent anaphylaxis by Fc domain targeting of FcγRIII
  di: Clay, Corey D., et al.
  Pubblicazione: (2017)
• Hypersensitivity reactions to asparaginase in mice are mediated by anti-asparaginase IgE and IgG and the immunoglobulin receptors FcεRI and FcγRIII
  di: Rathod, Sanjay, et al.
  Pubblicazione: (2019)
• Allergen-specific IgG1 and IgG3 through Fc gamma RII induce eosinophil degranulation.
  di: Kaneko, M, et al.
  Pubblicazione: (1995)
• Modulation of Immunoglobulin (Ig)E-mediated Systemic Anaphylaxis by Low-Affinity Fc Receptors for IgG
  di: Ujike, Azusa, et al.
  Pubblicazione: (1999)