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The BDNF Val66Met polymorphism enhances glutamatergic transmission but diminishes activity-dependent synaptic plasticity in the dorsolateral striatum

The Val66Met polymorphism in the brain-derived neurotrophic factor (BDNF) gene disrupts the activity-dependent release of BDNF, which might underlie its involvement in several neuropsychiatric disorders. Consistent with the potential role of regulated release of BDNF in synaptic functions, earlier s...

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Vydáno v:Neuropharmacology
Hlavní autoři: Jing, Deqiang, Lee, Francis S., Ninan, Ipe
Médium: Artigo
Jazyk:Inglês
Vydáno: 2016
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC5075499/
https://ncbi.nlm.nih.gov/pubmed/27378336
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuropharm.2016.06.030
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